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The purpose of this review was to critically evaluate the molecular basis of APAP-induced liver injury, with a focus on inflammasome signaling and emerging therapeutic interventions

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Generalized linear mixed effects models were used to compare the low versus normal carnitine status groups in patient measures of seizure frequency and severity, body mass index (BMI), number of antiseizure medications (ASM), beta-hydroxybutyrate (BHB), triglyceride, and carnitine levels across baseline, three to nine month follow-up (timepoint 1), one to two year follow-up (timepoint 2), and 2+ year follow-up (timepoint 3)