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In preclinical heart failure models, NNMT enzyme inhibition markedly reduced cardiac 1-MNA (by 77.1%), improved cardiac function, reduced left ventricular hypertrophy and fibrosis, and decreased pro-inflammatory gene expression
Without enough mitochondrial capacity to burn those fatty acids efficiently, they get recirculated and re-deposited, or worse, they overwhelm tissues that cant handle the load (a process called lipotoxicity)
It targets belly fat specifically while preserving muscle
Journal of Atherosclerosis Research
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