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In addition, selenium serves as a cofactor of glutathione peroxidase 4 (Gpx4) that inhibits lipid peroxidation, resulting in the attenuation of ferroptosis 17

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The modified intention-to-treat group consisted of 854 subjects

Several endogenous metabolites, including ubiquinol, vitamin E, and vitamin K hydroquinone, function as radical-trapping antioxidants that terminate lipid peroxyl radicals and suppress ferroptotic membrane damage 84-87