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Recently, numerous findings indicate that the defects of glucagon secretion get involve with development and exacerbation of hyperglycemia in T2D

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Dose adjustment, temporary treatment interruption, or switching to an alternative therapy may be appropriate

Furthermore, as energy substrates, SCFAs support mitochondrial respiration and enhance mitophagy to clear damaged mitochondria, reducing mtROS generation (49) a mechanism particularly relevant in the context of PCOS-associated mitochondrial dysfunction

Ive seen reports that this works for some people, but it will still require a physicians prescription

Preliminary lab work hints at improvements in inflammation and liver enzymes at sub-therapeutic dosesfindings Healthline notes remain early and anecdotal