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Because of this feedback mechanism, the administration of pharmacologic doses of exogenous thyroid hormone to patients with a normal thyroid suppresses endogenous thyroid hormone secretion

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With files from CBC's Pauline Dakin

A first hypothesis is that by attaching this insecticide to the agonist binding site, the nAChRs conformation is altered, blocking the open channel of the receptor and thus reducing the influence of Na + and Ca +2 ions on the nerve cell

Conversely, they have already been shown to promote production of the anti-inflammatory cytokine interleukin-10, which could exert a protective effect against noninfectious versions of the condition