TTC5 has previously been shown to downregulate ATP synthase activity, which sensitizes cells to mitochondrial-dependent apoptosis via enhanced p53 activity (72)
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This ludicrously harmful practice needs to be reversed
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Your body produces GHK-Cu on its own, but levels drop sharply with age
Im thinking, if you go too long without B12, or for whatever reason that month through stress/illness whatever you use up your B12 more quickly, when you do have your injection that your potassium could drop as your body goes through a healing process, similar to when you first go on it