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Zilka et al., 2017)

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In particular, the inhibition of eNOS results from the PKC phosphorylation of Thr495, amplified by the simultaneous dephosphorylation of Ser-1177 (Figure 1) ( Figure 1 2.3 Other players involved in endothelium-dependent vasodilatation and vasoconstriction Endothelium-derived contraction factors include Ang II, thrombin, thromboxane A2 (TxA2), O 2 , and endothelin 1 (ET-1) ( 2.4 Regulation of the endothelium integrity: the role of endothelial glycocalyx on NO bioavailability Macromolecules can cross the endothelial barrier through cellcell junctions, diffusion, vesicular transport, endothelial gaps, or through the cells themselves

In the absence of inflammation, basal levels of glucocorticoid receptor signaling promote the expression of PRRs, cytokine receptors, and complement factors, sensitizing cells to noxious stimuli and promoting the induction of inflammatory responses after tissue injury

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