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FIGURE 3 In summary, this evidence suggests that the origin of excess ROS production is the result of (1) defective mitochondria (due to the abnormal and reduced distribution of cardiolipins) with high intermembrane electrical potential and (2) reduced mitophagy process (due to high expression and activity of P53 protein)

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Indian J Microbiol 48(1):1934

The clinical translation process is slow

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The biggest risk factor for melanosis coli is frequent laxative use