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Evaluation of long acting GLP1R/GCGR agonist in a DIO and biopsy-confirmed mouse model of NASH suggest a beneficial role of GLP-1/glucagon agonism in NASH patients

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Mechanistic hypothesis: Dysesthesia is likely related to glucagon receptor activation, which retatrutide targets uniquely among obesity medications

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RNA sequencing data obtained from the DIO-MASH and CDA-HFD animal studies will be publicly available in the Gene Expression Omnibus ( under their respective data repository accession numbers: GSE294629 and GSE294630