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This elevation is evident in atherosclerosis (narrowing and hardening of arteries)

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Next, the time course of rebound in GSH levels after the CS exposure was examined

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Notably, GS, as a pivotal astrocytic enzyme, maintains glutamatergic-GABAergic equilibrium by converting synaptically released glutamate into glutamine, thereby preventing excitotoxicity and sustaining inhibitory GABA synthesis [42, 43]