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Nevertheless, the FA-SNCD@SBA-15/ENZ nanocarrier exhibited a rapid release of ENZ within a redox microenvironment, which can be attributed to the presence of disulfide bonds that cross-link the drug
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We hypothesize that iron dyshomeostasis is widespread in neurodegenerative diseases and ensuing mechanisms underlying iron toxicity may provide novel disease targets
Mogana Rajagopal, [email protected] This article was submitted to Ethnopharmacology, a section of the journal Frontiers in Pharmacology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
it does not incorporate patient genetic profiling or pharmacogenomic assessment into its platform recommendations