This interaction enables cells to transition smoothly from an initially acetylation-driven pro-inflammatory response to a lactylation-driven homeostatic repair gene expression, achieving precise temporal regulation of gene transcription patterns through metabolic flux (Hagihara et al., 2021)
10.1007/s40744-024-00723-9 118 NakayamaA.KurajohM.ToyodaY.TakadaT.IchidaK.MatsuoH
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