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Although free radicals can harm mitochondria, the mitochondria release many of them themselves during energy production (Lane, 2015

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The toxicity of free radicals contributes to the damage of cellular macromolecules (such as DNA, proteins and lipids), inflammation, tissue damage and subsequent cellular apoptosis (Reference Ames, Shigenaga and Hagen14)

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Importantly, many of the pathways susceptible to redox modulation are actively involved in the pathogenesis of ALD, including the NRF2 and AMPK pathways

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