HepG2 cells exhibited higher catalase activity than HeLa cells, and inhibiting catalase activity by using 3-aminotriazole (3-AT, a specific inhibition of catalase) or catalase siRNA remarkably reduced the resistance of HepG2 cells to ROS, demonstrating the key roles of catalase for the strong resistance of HepG2 cells to ROS
NGF and its receptors in the regulation of inflammatory response
10.1523/JNEUROSCI.0426-16.2016 10 AlamZ
Clinical trials showed joint pain rates similar to placebo, indicating no established direct causal relationship
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This is exciting research, but GLP-1 drugs are not a cure for OA
