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Inhibition of glycolysis pathway for the treatment of CRC The pronounced glycolysis dependency of CRC cells under normoxic conditions constitutes an exploitable metabolic vulnerability

Mammalian cell culture Human embryonic kidney 293T (ATCC, CRL-3216), U2OS (ATCC, HTB-96) and Vero (ATCC, CCL-81) cells were cultured in Dulbeccos modified Eagles medium Glutamax (DMEM

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102 stabilized GPX4 in intact HEK293T cells ( Figure 7 and Supplementary Figure S11 )