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However, L-carnitine may affect the body's reaction to the anti-arrhythmic effect of propafenone and mexiletine in patients with ischemia

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The characteristic brain lesions of cognitive dysfunction are neurofibrillary tangles (NFT) and senile plaques formed of amyloid- deposition, glycogen synthase kinase 3 beta (GSK3), and highly homologous kinase tau tubulin kinase 1 (TTBK1) can phosphorylate Tau proteins at different sites, overexpression of these enzymes produces extensive phosphorylation of Tau proteins making them insoluble and enhance NFT formation, which impairs cognitive functions

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