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Studies have shown the protective effects of Peroxiredoxin 1 (PRDX1) on stroke through disulfidptosis and the ischemic postconditionings (IPostC) mechanism

R162A and S353Y double mutant) were used to replace the EGFP gene, generating the AAV-hHPN WT and AAV-hHPN RS vectors

Substituting arginine for glutamate at this position markedly reduces the analogs affinity for the IGFBPs while leaving its ability to engage the type 1 IGF receptor (IGF-1R) largely intact