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This oxidative damage activates signaling pathways that promote inflammation and fibrosis, leading to glomerular hypertrophy, mesangial expansion, and tubulointerstitial fibrosis [183]

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It is hoped that tau-targeted ASOs may slow or halt neurodegeneration by preventing the aggregation of hyperphosphorylated tau into neurofibrillary tanglesbelieved to be a key driver of neuronal lossand the spreading of tau across neural networks (seeding)