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While targeting glutamine pathwaysparticularly GLS1 and -KG metabolismpresents promising antifibrotic potential, the cell type-specific and compensatory mechanisms highlight the need for precise therapeutic strategies to effectively harness glutamine-targeted interventions in IPF (Fig

Notably, CHAC2 displays significant structural differences in the loops surrounding its active site when compared to other GCTs

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Discussion 1) The study conducted by Chang et al aimed to identify the most effective drug for patients with recurrent glioma

Pottinger, A

Activation of the Nlrp3 inflammasome by mitochondrial reactive oxygen species: a novel mechanism of albumin-induced tubulointerstitial inflammation