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Regulation of potassium export channels by GSH is not yet proven in LAB and the mechanism for protection is suggested to be either sacrificial action of GSH which prevents rapid fall of intracellular pH or glutathionylation of glyceraldehyde 3-phosphate dehydrogenase (GAPDH) enzyme which helps to sustain glycolysis at the required level

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The 3.48% reduction in apoptotic cells did not reach statistical significance, though the molecular markers (caspase-3, PARP, Bcl-2/Bax) were all significant

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