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Moreover, CNS GIPR KO and mGIPR-Ab/GLP-1 treatment of wild-type mice both result in reduced activity in pathways associated with immune function, particularly neutrophil degranulation, suggesting that decreased CNS GIPR signalling might reduce obesity-associated inflammation 47,48,49

The issue is what happens when genuine interest in better energy, faster recovery, improved body composition, and higher quality of life gets met with hype instead of clinical guidance

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Not as a standalone intervention