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Importantly, integrating these imaging and omics-based approaches with GLP-1RA therapy may allow identification of patients with inflammation-driven or lipid-rich plaque phenotypes who are more likely to benefit from treatment

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Nitric oxide blocks cellular Heme insertion into a broad range of Heme proteins

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In effect, an oxidative intracellular environment may impair the function of Na + /H + antiporters, decreasing the intracellular pH and favoring an early phase of viral replication [84]