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Consequently, the dysregulation of BA metabolism in gout may stem from disruptions in BA biosynthesis, transport systems, and the composition of the gut microbiota

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The combined logic is: Fat mobilization (retatrutide) mitochondrial fat oxidation (MOTS-c via AMPK)

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The amylin receptor agonist class, which includes cagrilintide, adds a second mechanism of action that strengthens the weight loss effect