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Our central hypothesis is that CR activates metabolic reprograming pathways in DM defined by cancer hallmarks (e.g., uncontrolled cell proliferation, tumor suppressors loss etc.), while suppressing immune system surveillance

Future studies should focus on identifying its active constituents, elucidating detailed mechanisms of action, and validating its efficacy in well-designed clinical trials

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Researchers examining inflammatory disease models, gut epithelial biology, or mucosal immunity alongside tissue repair endpoints choose KLOW

L., Burguera, B., & Rothberg, M

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