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In fact, it acts on the production of cytokines, which are substances that influence the inflammatory processes in the body

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However, when the oxidant level exceeds the reducing abilities of cells, ROS can lead to cellular damage through the oxidation of biomolecules, such as proteins, lipids, and DNA [8]

In recent years, EAT has garnered extensive attention as a key risk factor for cardiovascular disease and a potential therapeutic target

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Alcoholic liver disease: Chronic alcohol consumption severely depletes hepatic glutathione through multiple mechanisms: increased ROS production during ethanol metabolism, reduced cysteine availability, and impaired mitochondrial glutathione transport