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In vitro , PYC exhibits antioxidant effects during adipogenesis by downregulating the mRNA expression of pro-oxidant enzymes, such as nicotinamide adenine dinucleotide phosphate hydrogen oxidase 4 and glucose-6-phosphate dehydrogenase, while simultaneously upregulating antioxidant enzymes, including Cu/Zn-SOD, Mn-SOD, GPx, and GSH reductase (Lee et al., 2012)

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598 FTH1 knockdown in neurons significantly inhibits cell viability and causes mitochondrial dysfunction, while overexpression of FTH1 impairs ferritinophagy, downregulates the expression of LC3 and NCOA4, and finally inhibits cell ferroptosis

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