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Comparing the glutathione-scaffold site among a variety of other members of the thioredoxin superfamily also provides an explanation for their disulfide substrate preferences, for example, why glutaredoxins prefer GSSR in contrast to thioredoxins lacking most of the relevant residues (Fig

In vivo isotope tracing reveals the versatility of glucose as a brown adipose tissue substrate

Mild glutathione synthetase deficiency usually results in the destruction of red blood cells (hemolytic anemia)

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