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The study confirmed that BPC-157s effects on tube formation were mediated via the ERK1/2 signalling pathway, as its effects were significantly inhibited by the ERK1/2 inhibitor PD98059

Interestingly, the elevated CHAC1 levels in TP53-mutant cells might represent an adaptive response, potentially compensating for defective ferroptosis pathways or preventing harmful GSH accumulation

20hr BSO: Cells treated with BSO and assayed for GSH after 20 hours

Enter: glutathione