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The described mechanism of L-carnitine action involves decrease of protease activity of the mitochondrial intramembrane cleaving protease PARL (presenilin-associated rhomboid-like protein), which mediates PINK1 cleavage by increasing interaction between PHB2 (Prohibitin-2) and PARL [144]

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Unlike synthetic cyanocobalamin, which the liver must first convert by removing a cyanide molecule and attaching a methyl group, methylcobalamin is already in its active, bioavailable state

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The autoimmune attack destroys cells in the stomach that produce a vital protein called intrinsic factor