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The family B1 GPCR: structural aspects and interaction with accessory proteins

Although our primary hypothesis concerns glutathione and glutamate, the underlying hypothesis is that in the residual state there is diminished neural metabolic integrity arising from a toxic process in the acute phase of illness [4]

Rarely, patients manifest disorientation, aphasia, grimacing, choreoathetoid movements, symptoms suggestive of encephalitis, episodes of hemiparesis or bulbar paralysis (318)

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