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While acute iron exposure produced minimal biochemical changes and did not sensitize cells to oxidative or ferroptotic challenges, chronic iron exposure induced ferritin upregulation, mitochondrial superoxide accumulation, suppression of GPX4 expression, elevated lipid peroxidation and loss of cellular glutathione (GSH)

Thymosin beta-4 plays a role in cell migration, blood vessel formation, wound healing, and inflammation control

Adoptive transfer of tumor-specific Th2 cells eradicates tumors by triggering an in situ inflammatory immune response

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Protective role of glutathione in the hippocampus after brain ischemia