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Indeed, the GSH and GSH-related enzymatic systems are efficient tools that cells have exploited in detoxification and, at the same time, represent the most ancient notice on the physiological role played by the tripeptide

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Studies have shown that RIPC can reduce myocardial infarction size and troponin levels by regulating calcium channel subunits, and it can also inhibit the NOX4-ROS signaling pathway to alleviate renal mitochondrial dysfunction, thereby lowering the risk of postoperative renal injury (178, 179)

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