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This mechanism establishes OLA as a promising anti-fibrotic therapeutic candidate ( Mitochondria-targeted antioxidants or CYP2E1 inhibition prevents this toxicity, highlighting CcO dysfunction as a key pathological mechanism ( Daphnia magna through its reactive metabolite NAPQI

Its expression can be induced by various cell types, such as endothelial cells, epithelial cells, and immune cells, under inflammatory stimuli (Tanaka et al., 2024)

Gastrointestinal Issues: This can cause constipation and diarrhea, generally when the dose is increased

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Overexpression of a transporter gene in a multidrug-resistant human lung cancer cell line