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Preclinical anabolic effect in catabolic states Tomas et al

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[1][7][9] Pathology Theory I theorize that KPs pathology can be explained by an insufficient bioavailability of IGF-1 and/or mutations in insulin receptors on keratinocytes

In prostate cancer, PGC-1 can promote tumor growth via androgen receptor activation while inhibiting invasion and metastasis through its interaction with estrogen-related receptor 46,47,48

Again, if there is an unexpected result, it is useful to repeat the test

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