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Although WPC administration caused weight gain in the MTX group, this increase was significantly lower than in the WPC-given control group (Table 2)

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Published mechanistic work points to BPC-157 enhancing VEGFR2 expression and downstream signalling activity in endothelial cells, rather than acting as a direct VEGFR2 ligand

The dense distribution of correlation signals across these immune compartments suggests that nicotine-related transcriptional changes may be involved in inflammatory cell recruitment, immune-cell activation, or immune imbalance in COPD ( Figure 2D )

In the digestive tract, BPC-157 stabilizes tight-junction proteins (e.g., occludin) and reduces oxidative stress, supporting mucosal integrity