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In the skeletal muscle of children with autism, skeletal abnormalities were concurrently found in those patients who had mitochondrial defects, which ranged from defects in complex I, III, IV, and V [56]

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Importantly, these alterations were also observed in idiopathic RBD (iRBD) patients, representing the prodromal stage of PD

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Upon activation, the tyrosine kinase activity of the IGF-IR entails sequential phosphorylation events at residues Y1135, Y1131, and Y1136, which produce conformational changes in the subunits to create docking sites for downstream signaling molecules and their phosphorylation (14)