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One reason may have to do with how trials measure the hormones effectiveness
Atrial remodeling: (1) Structural remodeling: Multiple factors, including inflammatory responses, paracrine/vasocrine effects of EAT, and elevated chronic hemodynamic stress, can activate atrial fibroblasts, promote their proliferation and differentiation, alter the expression of relevant proteins and cytokines, and ultimately drive the pathogenesis and progression of atrial fibrosis (4045)
Additionally, no significant change was observed in human studies, and the results were insufficient to draw any conclusions [28]
Moreover, the interplay between metabolism and the microenvironment and their relationship to tumor GSH levels has not been studied
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