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In the current study, we utilized the Bcl-2 inhibitor, HA14-1, to mimic loss of Bcl-2 function and assess the neuroprotective potential of Immunocal

As increased GABA-ergic neurotransmission has been implicated in neuronal inhibition associated with HE, the efficacy of utilizing benzodiazepine receptor antagonists to treat HE has been evaluated in several RCTs

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Henning Tidow for suggestions and manuscript proofreading

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Magnesium: While the MTHFR mutation does not cause magnesium deficiency, this mineral may help reduce cardiovascular risk in people with this gene mutation