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The FTD-like syndrome causing TREM2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism

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Conversely, the overexpression of Parkin resulted in a reduction in the number of neurons lost in the motor cortex of TDP-43 model animals while also inhibiting the degenerative phenotype observed in hTDP-43 model animals (Hebron et al., 2014)

Oncotarget .Read source Pickart L, Margolina A (2018)

Neuroprotection: Exploring the role of increased NAD+ and mitochondrial integrity in supporting cognitive focus and neuronal resilience