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The collagenase model replicates the matrix disorganization and degenerative pathology of clinical tendinopathy more closely than transection models, strengthening the relevance of the findings to the human chronic tendinopathy condition

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Kainate receptor post-translational modifications differentially regulate association with 4.1N to control activity-dependent receptor endocytosis

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In contrast, both hCiPSC-NSCs-treated rats (Fig

Some scientists are investigating whether BPC-157 could delay the aging process by promoting cell turnover and enhancing the body's ability to repair age-related damage in tissues and organs