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Its mechanism is fundamentally different MOTS-C acts at the level of cellular energy metabolism rather than extracellular matrix or tissue remodeling

In previous studies, renal or hepatic impairment had no clinically relevant effect on the pharmacokinetics of SC semaglutide [22, 23]

These findings were supported by the relaxation of large vessels (ex vivo), mainly by acting on vascular endothelial cells but also on vascular smooth muscle cells, increasing NO production and migration of endothelial cells, activating the signal pathways of Src, Cav-1, and eNOS, and reducing proteinprotein interaction between eNOS and Cav-1 [121,122]

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