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Central sensitization results from the noxious stimulus being transmitted to the secondary neurons in the trigeminal cervical complex via the afferent fibers of the trigeminal nerve

Complement Med

Interestingly, another mechanism was proposed as an alternative way to stabilize NRF2 through the interaction between the autophagy adaptor protein p62/sequestosome-1 (p62/SQSTM1) and KEAP1, which leads to the sequestration of KEAP1 in the autophagy-deficient cells and enables NRF2 accumulation [20]

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However, it is believed that common mechanisms underlying the behavioral deficits of ASD can be found [10]