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Previous studies have also demonstrated that GLP-1R agonists exendin-4 (Ex4) induced diuresis and natriuresis by increasing GFR and inhibiting the main renal proximal tubule sodium reabsorption in normotensive rats (101, 102)

In contrast, and consistent with data from mouse ventricular CMs, we observed that GLP-1(2836) failed to protect hESC-CMs from cytotoxicity under conditions of oxidative stress (Supplemental Figure 4C)