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Biomarker and Genetic Factors Influencing Concurrent Use Individual variation in oxidative stress response and antioxidant capacity is partly determined by genetic polymorphisms in pathways that regulate GLP-1 signaling, energy metabolism, and inflammation

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ALOX12 serves as the primary gene responsible for driving ferroptosis, and its mechanism of action involves the conversion of polyunsaturated fatty acids, particularly arachidonic acid, into the bioactive lipid (12S)-hydroperoxide eicosapentaenoic acid (Zheng et al., 2020)

Tryptophan degradation increases with stage in patients with rheumatoid arthritis