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Anti-inflammatory effects were also reported by the downregulation of neutrophil L-selectin (SELL) and elastase (ELANE), which are associated with neutrophil accumulation and the development of chronic skin wounds (Ashcroft et al., 1999), and reduction of infiltrating macrophages and promotion of macrophage polarization to the M2 (pro-repair) phenotype, ultimately leading to an accelerated transition from the inflammatory to the proliferative phase of healing (Campbell et al., 2014