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In this context, the concept of biased agonism 37 has emerged, in which ligands can selectively engage in different intracellular pathways, potentially allowing specific cellular responses

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2) suggests that BPC 157 administration prevents the chain of events after spinal cord injury that is mediated by the loss of local segmental inhibition and/or by an increased sensory afferent drive that results in the exacerbation of -motoneuron activity [66]

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