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Since IGFBP-4 acts predominantly by sequestering IGF-1, blocking its access to IGF1R, it is suggested that the tumorigenic actions of lnc-IGFBP4-1 are dependent on IGFBP-4 downregulation and IGF1R activation, but this remains to be definitively demonstrated
There is some evidence for a temporal window of TABLE 3-6 Examples of Experimental Models of Repetitive TBI vulnerability, with repeated mild injuries in close temporal proximity producing greater damage than injuries separated by longer periods of time (in the case of rodents, a separation of seven days) (Longhi et al., 2005)
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