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doi: 10.3389/fcimb.2024.1401963 134 ShuiLWangWXieMYeBLiXLiuYet al
CR in some experimental models enhances cell death rate, however, proliferation/ apoptosis rates vary through the course of treatment (5,11,21)
Knockout, knockdown, or pharmacologically inhibiting p53 boosted the anticancer efficacy of type I ferroptosis inducers (erastin and sulfasalazine)
Notably, xenobiotics including pharmaceutical residues and pesticides from urban and agro-industrial sources further complicate pollution profiles [31]
33 , 36 Recommendations for the treatment of adverse effects during acetylcysteine therapy have been proposed
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