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4B, D) revealed a significant inflammatory response in specific brain regions in untreated Pla2g6 / mice compared to age-matched wild-type controls (all p-values are shown in Supplementary Table S7)

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In addition, they support energy production in COCs and protect against oxidative stress-induced apoptosis

The pathophysiology of MASLD/MASH encompasses complex interactions among genetic background, impaired insulin signaling, hepatic lipid accumulation, inflammatory cascades, and progressive fibrosis

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