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Subsequent research revealed that PRMT6 is the primary catalyst responsible for this methylation, which exerts its effects by inhibiting gene expression through its interaction with H3 lysine 4 trimethylation (H3K4me3) and interfering with H3K4me3 reader binding [45,56]

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Writing in the journal Nature Medicine, a team led by Dr

The concern, while understandable from a mechanistic perspective, is not borne out by the evidence in human subjects

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